We seem to be bombarded with information about our diet nowadays and what gout foods should we eat and avoid if we suffer with this condition. Numerous articles in magazines and newspapers advise us to eat less fat (especially saturated fat of animals) and the amount of fruits and vegetables in our diet. Most of us also need to eat fewer calories and take more exercise to lose weight and increase our physical fitness, and this is especially true if we want to reduce pressure on the arthritic joints.
Gout is the only form of arthritis that symptoms can certainly be helped by means of diet. No expensive ingredients or nutritional supplements are required. Just cutting calories most people with primary gout can help, but the most important thing is to limit foods with high concentrations of purines. Some people report that gouty attacks are triggered by other foods (such as strawberries, citrus fruits, and tomatoes) that is not high in purines. If you are that the cause of these foods you have trouble notes, cut them out too.
How do we know that classical gout is connected with what we eat?
First, there is the clear historical proof of the middle-aged men, well fed gouty loved by cartoonists in the 17th and 18th centuries. A regular meal for these people consisted of different meat courses, washed down with abundant wine and spirits. On the other hand, although most people drank beer because of fear of contaminated drinking water, gout was not found in poorer people with a limited diet. Second, cases of primary gout very rare during the world wars of the 20th-century Europe, when food was rationed and very little meat was available. Such as food supplies returned to normal, and indeed as the population started eating better than ever before, gout (and obesity) was much more common in older men.
In the second half of the 20th century, a broad analysis of the chemical components of food and beverages is a direct link between a diet rich in purines and their role in triggering marked a gouty attack in susceptible people.
I read somewhere that Henry VIII had gout. Is this so?
The well-known portraits of an overweight middle-aged Henry VIII set sure he enjoyed food and drinks, both of which are risk factors for gout because they can lead to higher and higher levels of uric acid in the blood (hyperuricemia) over several years. Other risk factors include high blood pressure (hypertension) and Type 2 diabetes (the ' adult ' form), which we consider as diseases of abundance, as they are prevalent in the West. Most Western people eat a diet that today would have regarded as rich by Henry's topics; they could only dream of eating meat and drinking wine a day, but as long as their lean diet kept them alive, they were likely to suffer from gout. Hence gout known as the ' King of diseases and the disease of Kings '.
Answer to your question, Henry VIII had a number of problems – among them syphilis, which can lead to arthritis and school books say he had gout, too. Unfortunately for him, not us high • Urate levels to confirm the diagnosis testing!
The Prince Regent (who became King George IV) certainly had gout. He was racked with pain until in 1817, he began grateful use extracts of colchicum, an old remedy that recently accepted again by medical opinion (there's more gout remedies available here)
I've been on a bit of weight. Make over-weight is my gout symptoms worse?
If you are overweight, you should definitely try to cut the total amount of food you eat, to reduce the load on your joints, particularly on your knees and feet. A high body weight is the result of eating more than your body needs to continue, and the excess is stored as fat.
If you're overweight and have primary gout are various factors relevant. Overweight and obese people are at risk of developing high blood pressure and Type 2 diabetes, all of which can put extra stress on the kidneys and reduce the ability of the kidneys to excrete uric acid. Reducing your weight on the "healthy" range also helps lowering the concentration of uric acid in the plasma, causing the risks of crystals being deposited in the joints.
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